New acne viewpoint paper published

New Viewpoint Paper on Acne

An awesome new paper on current understanding of the causes of acne and how it can be treated was recently published in Experimental Dermatology (1). This is like a consensus paper that was written by 14 scientists from all over the world — USA, Japan, China, Germany, Taiwan, Hungary, Italy, South Korea, Lithuanina, and Switzerland. The paper is not yet in print and only available as an e-publication. It was published online on June 23 and you can find it here. Unfortunately, as for most scientific papers, you can only see the abstract for free. You will have to pay to get access to the whole paper as a PDF file.
Or you can just read all about it here at sanafela.com :-)

What’s in the paper?

The paper addresses pretty much the gammut of the current thinking about acne.
Some of the topics below may sound quite foreign to you, and I will do my best to explain it all in future posts. For now I just wanted to let you all know that this paper is out there, and then I’ll explain more as we go along.

  • Biology of sebaceous glands
  • Role of neuropeptides
  • Role of innate immunity
  • Stem cells for sebaceous glands
  • Properties of sebocytes
  • Role of hormones
  • Hyperkeratinization
  • Bacteria and sebum
  • Role of nutrition
  • Role of cytokines
  • Role of Toll-like receptors
  • New acne drugs
  • Plant extracts with anti-acne properties

References

(1) Kurokawa et al (2009) New developments in our understanding of acne pathogenesis and treatment. Exp Dermatol. 2009 Jun 23. [Epub ahead of print]

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This entry was posted in acne, prevention, research, treatments. Bookmark the permalink. Post a comment or leave a trackback: Trackback URL.

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  1. [...] As promised, here is a follow-up post to the acne viewpoint paper that was published in Experimental Dermatology (Ref 1) . This paper was published very recently and summarizes the state-of-the-art of our knowledge about acne. This post explains recent insights in the way sebocytes recognize and respond to bacteria. [...]

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